Karl
Note: This article below is a very
orthodox description of the kidney, and one
particular kidney disease. There is no
question in my mind, although I am certainly
not a doctor, that the original damage to the
kidney, described below, is actually caused
by the activity of free radicals hitting the
kidney. The report below admits that
the disease is of "uncertain origin."
There is more and more acceptance of the
concept that free radicals are the SOLE cause
of all degenerative diseases, including heart
disease and cancer.
Free radicals are greatly increased in number and activity by the presence of heavy metals in the body. There is no possible way to remove these free radicals except through chelation therapy -- either intravenous or oral chelation.
In many cases the original damage can be reversed and the parts of the kidney that have been damaged can be revitalized simply by discontinuing the bombardment of the area by free radicals.
Thus, if you remove the metals from the body, with chelation therapy, and IF the damage to the kidney can be reversed, then the elimination of these metals would likely have a very positive effect on the kidney.
In other words, membranous nephropathy could be treated with a less invasive and less dangerous approach than drugs. As mentioned below, this disease is caused by the "thickening of the walls" of the capillaries. That is rather similar to the blockage of the larger vessels, called arteries. If chelation can improve blood flow in the arteries, which it can, then it would be reasonable to conclude that it should improve blood flow also in the capillaries.
One of the traditional tests is a biopsy of the kidney. I would think that no person would want to have a biopsy without trying, first, less invasive and dangerous tests. One test would certainly be a 60 day period of chelation therapy. This would be a test to see if the symptoms reduced. Since, by law, only drugs are allowed to "cure" a disease, it would be clear that chelation therapy would not be "curing" this problem, but simply removing metals from the body, and thereby allowing the body to heal or "cure" itself by the natural processes when the body is no longer being attacked by so many free radicals.
It certainly seems worth a try, does it not??
Karl Loren
Kidney anatomy
The kidneys are responsible for removing wastes from the body, regulating electrolyte balance and blood pressure, and the stimulation of red blood cell production.
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Alternative names
Return to topMembranous glomerulonephritis; Membranous GN; Extramembranous glomerulonephritis; Glomerulonephritis - membranous
A kidney disorder resulting in disruption of kidney function because of inflammation of the glomerulus and changes in the glomerular basement membrane.
Causes and risks Return to top
The glomeruli are the inner
structures of the kidney that include small capillaries
surrounded by membranes through which the blood is
filtered to form urine. Membranous nephropathy is caused
by thickening of the capillary wall of the glomerular
basement membrane (the deepest membrane) by immune
complexes. The cause is not known.
It is one of the most common causes of
nephrotic syndrome, which is the most common
presentation of the disease. It may also appear as
asymptomatic excretion of
protein in the urine. Glomerular filtration rate (the
"speed" of blood purification) is usually nearly normal,
and examination of sediment in the urine may be
unremarkable or may show oval fat bodies, and hyaline,
granular, and
fatty casts.
Membranous nephropathy may be a primary
renal disease of uncertain origin, or it may be
associated with other conditions. Risks include
systemic disorders such as
Hepatitis B,
malaria, malignant solid tumors,
non-Hodgkin’s lymphoma,
systemic lupus erythematosus,
syphilis, and others. Risks also include exposure to
substances or medications, including gold,
mercury, penicillamine, trimethadione,
skin-lightening creams, and others.
The disorder occurs in approximately 2 out of 10,000
people. It may occur at any age but is more common after
age 40.
Prompt treatment of associated disorders, and avoidance of associated substances, may reduce risk.
Note: Symptoms vary and no symptoms may be present in many cases.
An examination may be nonspecific except for edema. A urinalysis may reveal protein in the urine and/or blood in the urine (hematuria). Serum albumin may be low. Blood lipid levels may increase. Kidney biopsy confirms the diagnosis of membranous nephropathy.
The goal of treatment is to minimize symptoms and slow
the progression of the disease. Symptoms should be
treated as appropriate. Medications vary. Often,
corticosteroids or immunosuppressive medications may be
used to attempt to reduce symptoms and progression of the
disorder, with variable results. Medications to treat
symptoms may include antihypertensive and diuretic
medications. Antibiotics may be needed to control
infections.
Treatment of
high blood cholesterol and triglyceride levels is
recommended to reduce the development of
atherosclerosis secondary to
nephrotic syndrome. Dietary limitation of
cholesterol and saturated
fats may be of only limited benefit as the high
levels of cholesterol and triglyceride seem to be caused
by overproduction by the liver rather than excessive
intake of fats. Medications to reduce cholesterol and
triglycerides may be recommended.
Karl Note:
Any thought that "high cholesterol" is a sign of any
disease is false.
Click here to read my report on the truth about
cholesterol lowering drugs.]
Affected individuals are at increased risk for thrombotic
(clotting) events involving the lungs (pulmonary
embolisms) and legs (deep venous thromboses often
referred to as DVTs). Those affected are therefore
occasionally prescribed aspirin or other blood thinners
to prevent these complications.
High-protein diets are of debatable value. In many patients, reducing the amount of protein in the diet produces decrease in urine protein. In most cases, a moderate protein diet (1 gram of protein per kilogram of body weight per day) is usually recommended. Sodium in diet (salt) may be restricted to help control edema. Vitamin D may need to be replaced if nephrotic syndrome is chronic and unresponsive to therapy.
Remissions and exacerbations may occur with or without therapy. The course of the disorder is highly variable. Spontaneous remission is possible, as is a variable course of remissions (symptom-free periods) and acute symptomatic episodes. 70 to 90% will have some degree of irreversible kidney damage within 2 to 20 years, with about 20% progressing to end-stage renal disease.
Call your health care provider if Return to top
Call for an appointment with your health care provider if symptoms indicate membranous nephropathy may be present. Call for an appointment with your health care provider if symptoms worsen or persist, if you experience a decreased urine output or other new symptom develops.
Update Date: 11/30/01
Updated by: Andrew Koren, M.D.,
Department of Nephrology, NYU-Mount Sinai Medical Center,
New York, NY. Review provided by VeriMed Healthcare
Network.
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