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Alternative names
Return to topMembranous glomerulonephritis; Membranous GN; Extramembranous glomerulonephritis; Glomerulonephritis - membranous
A kidney disorder resulting in disruption of kidney function because of inflammation of the glomerulus and changes in the glomerular basement membrane.
Causes and risks Return to top
The glomeruli are the inner structures of the
kidney that include small capillaries surrounded by membranes through which the
blood is filtered to form urine. Membranous nephropathy is caused by thickening
of the capillary wall of the glomerular basement membrane (the deepest membrane)
by immune complexes. The cause is not known.
It is one of the most common causes of
nephrotic
syndrome, which is the most common presentation of the disease. It may also
appear as
asymptomatic excretion of
protein in
the urine. Glomerular filtration rate (the "speed" of blood purification) is
usually nearly normal, and examination of sediment in the urine may be
unremarkable or may show oval fat bodies, and hyaline, granular, and
fatty casts.
Membranous nephropathy may be a primary
renal
disease of uncertain origin, or it may be associated with other conditions.
Risks include
systemic
disorders such as
Hepatitis B,
malaria,
malignant solid tumors,
non-Hodgkin's lymphoma,
systemic
lupus erythematosus,
syphilis,
and others. Risks also include exposure to substances or medications, including
gold,
mercury, penicillamine, trimethadione, skin-lightening creams, and others.
The disorder occurs in approximately 2 out of 10,000 people. It may occur at any
age but is more common after age 40.
Prompt treatment of associated disorders, and avoidance of associated substances, may reduce risk.
Note: Symptoms vary and no symptoms may be present in many cases.
An examination may be nonspecific except for edema. A urinalysis may reveal protein in the urine and/or blood in the urine (hematuria). Serum albumin may be low. Blood lipid levels may increase. Kidney biopsy confirms the diagnosis of membranous nephropathy.
The goal of treatment is to minimize symptoms and slow the progression of the disease. Symptoms should be treated as appropriate. Medications vary. Often, corticosteroids or immunosuppressive medications may be used to attempt to reduce symptoms and progression of the disorder, with variable results. Medications to treat symptoms may include antihypertensive and diuretic medications. Antibiotics may be needed to control infections.
[Karl Note: These would be exactly the
wrong treatments. Also, avoid any anti-depressants.
"Antihypertensive" drugs are often psychiatric drugs -- the most harmful of any
class of drugs. The sequence of remedy would be, first, examine the
wrong
relationships in the person's life, next look at any need for
detoxifying the
body, next
change the diet, and finally, after all these others, take my
oral chelation
formula.
Treatment of
high blood
cholesterol and triglyceride levels is recommended to reduce the development
of
atherosclerosis secondary to
nephrotic
syndrome. Dietary limitation of
cholesterol
and saturated
fats
may be of only limited benefit as the high levels of cholesterol and
triglyceride seem to be caused by overproduction by the liver rather than
excessive intake of fats. Medications to reduce cholesterol and
triglycerides may be recommended.
[Karl Note: This is the
to-be-expected false remedy.
Click here
to read the truth about so-called high cholesterol.]
Affected individuals are at increased risk for thrombotic (clotting) events
involving the lungs (pulmonary embolisms) and legs (deep venous thromboses often
referred to as DVTs). Those affected are therefore occasionally prescribed
aspirin or other blood thinners to prevent these complications.
[Karl Note: Blood thinners, like Coumadin, are terrible drugs. Avoid them! Click Here for my article about blood thinners.]
High-protein diets are of debatable value. In many patients, reducing the amount of protein in the diet produces decrease in urine protein. In most cases, a moderate protein diet (1 gram of protein per kilogram of body weight per day) is usually recommended. Sodium in diet (salt) may be restricted to help control edema. Vitamin D may need to be replaced if nephrotic syndrome is chronic and unresponsive to therapy.
[Karl Note: More false information. In fact an increase of protein, best in raw, unprocessed form, is helpful. Click here for my diet recommendations.]
Remissions and exacerbations may occur with or without therapy. The course of the disorder is highly variable. Spontaneous remission is possible, as is a variable course of remissions (symptom-free periods) and acutesymptomatic episodes. 70 to 90% will have some degree of irreversible kidney damage within 2 to 20 years, with about 20% progressing to end-stage renal disease.
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